
Publications
Scientific articles and outputs from PGNM teams and collaborators.
About
PGNM publications reflect the scientific output of the laboratory’s teams and collaborators, spanning fundamental mechanisms, disease models, technologies and translational research.
2016
Phosphatidylinositol 3-kinase inhibition restores Ca2+ release defects and prolongs survival in myotubularin-deficient mice.,
Proc Natl Acad Sci U S A 2016 Dec; 113(50): 14432-14437.
2016
Macrophage PPARγ, a Lipid Activated Transcription Factor Controls the Growth Factor GDF3 and Skeletal Muscle Regeneration.,
Immunity 2016 Nov; 45(5): 1038-1051.
2016
The interaction between herpes simplex virus 1 genome and promyelocytic leukemia nuclear bodies (PML-NBs) as a hallmark of the entry in latency,
Microb Cell 2016 Nov; 3(11): 569-572.
2016
[NeuroMyoGene Institute: a Franco-Canadian partnership promoting research in neuromuscular disorders].,
Med Sci (Paris) 2016 Nov; 32 Hors série n°2(): 55-56.
2016
Real-Time Tracking of Parental Histones Reveals Their Contribution to Chromatin Integrity Following DNA Damage.,
Mol Cell 2016 Oct; 64(1): 65-78.
2016
A transgenic mouse expressing CHMP2Bintron5 mutant in neurons develops histological and behavioural features of amyotrophic lateral sclerosis and frontotemporal dementia.,
Hum Mol Genet 2016 Aug; 25(15): 3341-3360.
2016
mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis.,
J Mol Cell Cardiol 2016 Aug; 97(): 213-25.
2016
Maurocalcin phosphorylated at threonin 26 maintains its activity on ryanodine receptor-mediated Ca2+ release in intact muscle fibers.,
Proc Natl Acad Sci U S A 2016 Jul; 113(30): E4264-5.
2016
Time-lapse scanning surface plasmon microscopy of living adherent cells with a radially polarized beam.,
Appl Opt 2016 Feb; 55(6): 1216-27.
2016
Latency Entry of Herpes Simplex Virus 1 Is Determined by the Interaction of Its Genome with the Nuclear Environment,
PLoS Pathog 2016 Sep; 12(9): e1005834.
2016
Notch Stimulates Both Self-Renewal and Lineage Plasticity in a Subset of Murine CD9High Committed Megakaryocytic Progenitors.,
PLoS One 2016 ; 11(4): e0153860.