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The TUDOR domain of SMN is an H3K79 me1 histone mark reader
Interplay between PML NBs and HIRA for H3.3 dynamics following type I interferon stimulus
Team: Fundamental and pathophysiological mechanisms implicated in ataxia
Publication: Recent Advances on Therapeutic Approaches for Friedreich’s Ataxia: New Pharmacological Targets, Protein, and Gene Therapy
Hélène Puccio
